Supportive Periodontal Therapy
Dental Procedures
≈ ¥200-600
(≈ $30-90)
30 min
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Description
Estimated cost for Supportive Periodontal Therapy at general public hospitals in China is about ¥140-480, and at Grade 3A hospitals about ¥200-600, varying by hospital tier and region.
Main Uses
SPT is the cornerstone of post-active periodontal therapy care. Its primary purposes are: preventing recurrence/progression of periodontitis, maintaining achieved periodontal health and tooth/implant longevity, controlling subgingival biofilm and inflammation, enabling early detection of disease reactivation or new lesions, and supporting patients with systemic risk factors (e.g., diabetes, cardiovascular disease). It is indicated for all patients with a history of chronic or aggressive periodontitis, peri-implant mucositis/peri-implantitis, or significant periodontal risk factors.
Normal Range
Periodontal Maintenance Therapy (SPT) is not a laboratory test with numerical 'normal ranges'; it is a clinical, non-invasive, long-term care protocol. Key clinical parameters monitored during SPT include: probing depth (PD) ≤3 mm, clinical attachment level (CAL) stable (no further loss), bleeding on probing (BOP) <10–15% of sites, plaque control record (PCR) <20–30%, no suppuration, and radiographic bone level stability. These reflect periodontal health maintenance—not quantitative lab values.
Low Values - Possible Causes
N/A — SPT is a therapeutic procedure, not a measurable biomarker test; thus, 'low values' do not apply. However, suboptimal outcomes (e.g., high BOP%, deep PDs, or CAL loss) may result from: inadequate patient oral hygiene compliance, infrequent SPT intervals (>3–4 months), undiagnosed systemic conditions (e.g., uncontrolled diabetes), smoking, or residual subgingival calculus/biofilm.
High Values - Possible Causes
N/A — SPT has no 'high values'. Clinical deterioration (e.g., elevated PD, increased BOP%, progressive CAL loss) may be caused by: poor adherence to maintenance schedule, persistent microbial dysbiosis, untreated peri-implantitis, genetic susceptibility (e.g., IL-1 polymorphism), or medication-induced gingival overgrowth (e.g., phenytoin, nifedipine).