吲哚青绿血管造影（ICGA）
眼科项目
≈ ¥300-800
(≈ $40-110)
大约20-40分钟（含准备与注射，图像采集约5-10分钟，报告通常2-4小时出具）
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项目介绍
吲哚青绿血管造影（ICGA）在一般公立医院参考费用约¥210-640，三甲医院参考费用约¥300-800，常用于相关诊断评估与就医安排，费用随医院及地区有所差异。
主要用途
ICGA is primarily used to evaluate choroidal vasculature and pathology when standard fluorescein angiography (FA) is insufficient. Key clinical applications include: diagnosing and monitoring polypoidal choroidal vasculopathy (PCV); differentiating types of choroidal neovascularization (CNV); evaluating chronic central serous chorioretinopathy (CSC); detecting occult or inflammatory choroidal lesions (e.g., in Vogt-Koyanagi-Harada disease or sarcoidosis); assessing choroidal tumors (e.g., choroidal melanoma vs. hemangioma); and guiding photodynamic therapy (PDT) planning for CNV or PCV.
正常值范围
ICGA is a qualitative/semi-quantitative imaging test—not a blood assay with numerical 'values'. It assesses choroidal vascular dynamics via real-time near-infrared fluorescence. Normal findings include: rapid choroidal filling (within 10–15 seconds), homogeneous and intense choroidal fluorescence, well-defined choroidal vessel architecture, no leakage or pooling, and clearance of dye from the choroid by 15–30 minutes. No standardized quantitative 'normal range' exists for serum ICG concentration; plasma half-life is ~3–4 minutes, and >98% protein-bound; serum ICG levels are not clinically measured.
偏低可能原因
Low or absent choroidal fluorescence on ICGA may result from: 1) Severe choroidal hypoperfusion (e.g., in giant cell arteritis or severe hypotension); 2) Choroidal atrophy (e.g., advanced age-related choroidal thinning or choroideremia); 3) Massive subretinal hemorrhage or exudate blocking ICG signal; 4) Technical factors (e.g., incorrect dye injection timing, underdosing <0.5 mg/kg, poor camera alignment, or filter malfunction); 5) Severe hepatic dysfunction impairing ICG clearance (rarely affects early-phase imaging but may reduce peak fluorescence intensity).
偏高可能原因
Increased or prolonged choroidal fluorescence on ICGA may be caused by: 1) Choroidal hyperperfusion (e.g., in inflammatory conditions like posterior scleritis or VKH syndrome); 2) Choroidal vascular leakage due to neovascularization (e.g., polypoidal choroidal vasculopathy or choroidal neovascularization); 3) Stasis or delayed drainage (e.g., in central serous chorioretinopathy—CSC—with dilated choroidal vessels); 4) Dye recirculation artifacts or overdosing (>0.7 mg/kg); 5) Reduced hepatic metabolism (e.g., cirrhosis or Gilbert syndrome), leading to prolonged systemic ICG retention and enhanced late-phase background fluorescence.